Showing posts with label lower. Show all posts
Showing posts with label lower. Show all posts
Research shows the more chocolate you eat the lower your body fat level
Monday, May 19, 2014
University of Granada researchers from the Faculty of Medicine and the Faculty of Physical Activity and Sports Sciences have scientifically disproven the old belief that eating chocolate is fattening. In an article published this week in the journal Nutrition, the authors have shown that higher consumption of chocolate is associated with lower levels of total fat (fat deposited all over the body) and central fat (abdominal), independently of whether or not the individual participates in regular physical activity and of diet, among other factors.
The researchers determined whether greater chocolate consumption associated with higher body mass index and other indicators of total and central body fat in adolescents participating in the HELENA (Healthy Lifestyle in Europe by Nutrition in Adolescence) study. This project, financed by the European Union, studies eating habits and lifestyle in young people in 9 European countries, including Spain.
Independent of diet and physical activity
The study involved 1458 adolescents aged between 12 and 17 years and results showed that a higher level of chocolate consumption associated with lower levels of total and central fat when these were estimated through body mass index, body fat percentage—measured by both skinfolds and bioelectrical impedance analysis—and waist circumference. These results were independent of the participants sex, age, sexual maturation, total energy intake, intake of saturated fats, fruit and vegetables, consumption of tea and coffee, and physical activity.
As the principle author Magdalena Cuenca-GarcĂa explains, although chocolate is considered a high energy content food—it is rich in sugars and saturated fats—“recent studies in adults suggest chocolate consumption is associated with a lower risk of cardiometabolic disorders”.
In fact, chocolate is rich in flavonoids—especially catechins—which have many healthy properties: “they have important antioxidant, antithrombotic, anti-inflammatory and antihypertensive effects and can help prevent ischemic heart disease”.
Recently, another cross-sectional study in adults conducted by University of California researchers found that more frequent chocolate consumption also associated with a lower body mass index. Whats more, these results were confirmed in a longitudinal study in women who followed a catechin-rich diet.
The effect could be partly due to the influence of catechins on cortisol production and on insulin sensitivity, both of which are related with overweight and obesity.
Calorie impact is not the only thing that matters
The University of Granada researchers have sought to go further and analyse the effect of chocolate consumption at a critical age like adolescence by also controlling other factors that could influence the accumulation of fat. The research, which is both novel and, perhaps, the largest and best-controlled study to date, is the first to focus on the adolescent population. It includes a large number of body measures, objective measurement of physical activity, detailed dietary recall with 2 non-consecutive 24-hour registers using image-based software, and controls for the possible effect of a group of key variables.
In Nutrition, the authors stress that the biological impact of foods should not be evaluated solely in terms of calories. “The most recent epidemiologic research focuses on studying the relation between specific foods—both for their calorie content and for their components—and the risk factors for developing chronic illnesses, including overweight and obesity”.
Despite their results, the authors insist that chocolate consumption should always be moderate. “In moderate quantities, chocolate can be good for you, as our study has shown. But, undoubtedly, excessive consumption is prejudicial. As they say: you can have too much of a good thing”.
The University of Granada researchers stress that their findings “are also important from a clinical perspective since they contribute to our understanding of the factors underlying the control and maintenance of optimal weight”.
Delaying retirement could lower the risk of brain disease in the elderly
Sunday, May 11, 2014
Delaying retirement could lower the risk of brain disease in the elderly - Recent research has shown that people who delayed retirement have a lower risk of brain diseases such as Alzheimers and dementia compared with those who retired at the age of 60 years.
The research was conducted on 429,000 employees in France. They found that the risk of dementia decreased when somebody delay their retirement and keep working at the age of 60 years and over.
"Our data show strong evidence for a decreased risk of dementia and brain disease associated with old age when one is put off retirement and continue working in old age," said Carole Dufoil researchers from INSERM, as reported by Reuters.
INSERM found that there is a reduced risk of Alzheimers by 14 percent for those who retire at age 65 years, compared with those who retired at the age of 60 years. The results obtained through observations of the participants for 12 years and ended in 2010.
Dean Hartley, director of the Alzheimers Association explains that the physical and intellectual activities undertaken by the elderly to help them reduce the risk of brain diseases such as Alzheimers and dementia. Even so Hartley argued that more research is needed to determine the exact reason behind the link between pensions and the risk of brain disease.
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The research was conducted on 429,000 employees in France. They found that the risk of dementia decreased when somebody delay their retirement and keep working at the age of 60 years and over.
"Our data show strong evidence for a decreased risk of dementia and brain disease associated with old age when one is put off retirement and continue working in old age," said Carole Dufoil researchers from INSERM, as reported by Reuters.
INSERM found that there is a reduced risk of Alzheimers by 14 percent for those who retire at age 65 years, compared with those who retired at the age of 60 years. The results obtained through observations of the participants for 12 years and ended in 2010.
Dean Hartley, director of the Alzheimers Association explains that the physical and intellectual activities undertaken by the elderly to help them reduce the risk of brain diseases such as Alzheimers and dementia. Even so Hartley argued that more research is needed to determine the exact reason behind the link between pensions and the risk of brain disease.
Vitamin D3 Halts Inflammation to Lower Cardiovascular Disease Risk
Tuesday, May 6, 2014

Thousands of research studies have demonstrated the powerful health-promoting properties of the prohormone compound, vitamin D3. Researchers publishingin The Journal of Immunology explain the specific molecular and signaling events by which vitamin D inhibits inflammation in the human body. In a very detailed analysis, scientists show that low blood circulating levels of vitamin D do not adequately inhibit the inflammatory cascade necessary to turn off this potentially destructive mechanism.
Low levels of inflammation, normally used by the body to fight pathogenic invaders, become a primary cause of heart disease, diabetes, many cancer lines and Alzheimer’s dementia. Additional evidence reportedin the British Journal of Nutrition demonstrates that adequate vitamin D levels reduce critical markers of cardiovascular health and can lead to significant reductions in body fat in overweight and obese people. It is critical to have your vitamin D level checked, and supplement as necessary to lower inflammation levels and risk of heart disease.
Vitamin D Lowers Inflammation to Prevent Chronic Disease and Stimulate Immune Response Systems

Dr. Elena Goleva, lead scientist from National Jewish Health, found that prior studies with vitamin D show a clear and positive link between blood levels of the prohormone and a variety of different health outcomes. Dr. Goleva and her team looked for specific mechanisms to explain precisely how vitamin D functions at the cellular level to prevent disease in the human body.
Dr. Goleva and her team of researchers noted that vitamin D is a catalyst that initiates “a clear chain of cellular events, from the binding of DNA, through a specific signaling pathway, to the reduction of proteins known to trigger inflammation.” To test how vitamin D acts on immune and inflammatory pathways, team scientists exposed human white blood cells to varying levels of vitamin D and exposed them to an agent known to promote intense inflammatory responses and advance disease processes.
Vitamin D Lowers Inflammatory Markers and Increases Health-Promoting HDL Cholesterol

White blood cells that were incubated with no vitamin D or a solution of 15 ng/mL produced very high levels of the pro-inflammatory cytokines IL-6 and TNF-alpha. In vivo, these signaling messengers are known to be responsible for the development and progression of cardiovascular disease, certain forms of cancer and dementia. Cells incubated at a concentration of 30 ng/mL and above showed a significantly reduced response, while the highest level of inflammatory inhibition occurred at 50 ng/ml.
Supporting evidence for the health-promoting effect of vitamin D as reported in the journal BMJ shows marked improvement in cardiovascular biomarkers including HDL cholesterol. Additionally, researchers found the hormone-like substance lowered the risk of lipid peroxidation, the process responsible for making LDL cholesterol molecules sticky and prone to form atherosclerotic plaque. They also determined that high circulating levels of vitamin D were associatedwith “significant reductions in fat mass”in overweight and obese people.
There should be no doubt that the millions of people living with grossly sub-optimal vitamin D levels are dramatically increasing their risk of developing a plethora of potentially deadly illnesses. Health-minded individuals will ensure they maintain a circulating blood level of 50 to 70 ng/mL by means of a 25(OH)D blood test to regulate systemic inflammation and fight chronic disease.
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